Fetal Cell Lines in Vaccines & Autism
Issues Law Med. 2015 Spring;30(1):47-70.
Epidemiologic and Molecular Relationship Between Vaccine Manufacture and Autism Spectrum Disorder Prevalence.
The concern being addressed (after the documentation of human DNA, at least in fragmented form) is whether or not such “vaccine DNA” could be incorporated into the living “human DNA” of the host recipient; however, that is only one rather minor concern with the injection of human DNA from one person’s cells into the body of another living human being. From an immunology and inflammology standpoint, the concern is the likelihood that exogenous human DNA would trigger inflammatory responses after being perceived by the immune system and received by receptors for DAMPs (damage associated molecular patterns). Whether this amount of inflammation triggered from human DNA in vaccines is great or small, frequent or uncommon is unknown due to the lack of adequate vaccine testing; however, one would expect it to be of greater consequence in “genetically susceptible” persons and also when co-administered in various mixtures with other vaccines and with their pro-inflammatory ingredients such as aluminum (which exacerbates the release of human DNA), mercury (known to promote immune sensitization) and allergenic antibiotics common in vaccines, including streptomycin, polymyxin B, neomycin, gentamicin and kanamycin.
 "Although DNA DAMPs are closely associated with the development of autoimmune disease, DNA DAMPs also contribute to the activation of acquired immune responses following vaccination with alum adjuvant. Previous studies have shown that genomic DNA from dying cells induces the maturation of antigen-presenting cells as well as antigen-specific antibody and cytotoxic T cell responses. This suggests that self-DNA DAMPs can activate innate immune responses that induce acquired immunoresponses. Recently, Marichal et al. demonstrated that the adjuvanticity of alum was dependent on self-DNA released from cells at the alum inoculation site (Marichal et al., 2011). NLRP3 appears to be a key sensor in the induction of alum-mediated innate immunity, although its function is only partially dependent upon alum adjuvanticity. " Jounai et al. Recognition of damage-associated molecular patterns related to nucleic acids during inflammation and vaccination. Front Cell Infect Microbiol. 2013 Jan 8;2:168  Romano et al. Anaphylaxis to streptomycin. Allergy. 2002 Nov;57(11):1087-8  Henao MP, Ghaffari G. Anaphylaxis to polymyxin B-trimethoprim eye drops. Ann Allergy Asthma Immunol. 2016 Apr;116(4):372  Goh CL. Anaphylaxis from topical neomycin and bacitracin. Australas J Dermatol. 1986 Dec;27(3):125-6  Sánchez-Pérez et al. Allergic contact dermatitis from gentamicin in eyedrops, with cross-reactivity to kanamycin but not neomycin. Contact Dermatitis. 2001 Jan;44(1):54